The Relationship between Self-Control in Adolescence and Social Consequences in Adulthood: Assessing the Influence of Genetic Confounds

Purpose

Assess the relationship between levels of self-control in adolescence and a variety of later-life outcomes and evaluate the confounding effects of genetic factors.

Methods

The current study employed data from the National Longitudinal Study of Adolescent Health (Add Health) and examined whether levels of self-control in adolescence are related to economic, educational, employment, health, relationship and family, and behavioral outcomes in adulthood using DeFries-Fulker regression-based analyses.

Results

Analyses employing non-genetically sensitive methods indicated robust associations between self-control and various social consequences. After estimating genetically-sensitive analyses, however, many associations were no longer significant. Those associations which remained significant were in the reversed direction relative to the non-genetically sensitive models. Additionally, further analyses indicated that some of the remaining significant associations were influenced by nonshared genetic effects.

Conclusions

The findings indicate that even after controlling for the effect of genetic factors, levels of self-control are associated with differences in a variety of social outcomes. However, given the reduction in the number of significant associations and reversal of associations in the genetically sensitive models, analyses of the social consequences of low self-control which do not account for the effect of genetic factors are likely misspecified.     

Genetic and environmental contributions to stability and change in levels of self-control

Purpose

There has been an emerging body of research estimating the stability in levels of self-control across different sections of the life course. At the same time, some of this research has attempted to examine the factors that account for both stability and change in levels of self-control. Missing from much of this research is a concerted focus on the genetic and environmental architecture of stability and change in self-control.

Methods

The current study was designed to address this issue by analyzing a sample of kinship pairs drawn from the Child and Young Adult Supplement of the National Longitudinal Survey of Youth 1979 (CNLSY).

Results

Analyses of these data revealed that genetic factors accounted for between 74 and 92 percent of the stability in self-control and between 78 and 89 percent of the change in self-control. Shared and nonshared environmental factors explained the rest of the stability and change in levels of self-control.

Conclusions

A combination of genetic and environmental influences is responsible for the stability and change in levels of self-control over time.     

The influence of neuropsychological deficits in early childhood on low self-control and misconduct through early adolescence

Purpose

Gottfredson and Hirschi (1990) contend that low self-control is the result of parental management techniques. However, an emerging line of research has revealed that neuropsychological deficits influence the development of low self-control ( and ). Nevertheless, these studies have largely tested the effects of neuropsychological deficits on low self-control cross-sectionally or in the short term. This study addresses an important void in the literature by examining the influence of neuropsychological deficits in early childhood on levels of self-control and misconduct through early adolescence.

Methods

Data come from the Early Childhood Longitudinal Survey, Kindergarten (ECLS-K), the largest nationally representative sample of U.S. children.

Results

We found that deficits in neuropsychological functioning during kindergarten were consistently predictive of lower levels of self-control during the third, fifth, and eighth grade as well as higher levels of conduct problems during the eighth grade. These effects remained significant after accounting for demographic variables, features of the neighborhood, and a number of parenting variables.

Conclusions

Neuropsychological deficits during early childhood play an important role in the development of low self-control through early adolescence and misconduct during early adolescence.     

Stressful life events and adolescent drug use: Moderating influences of the MAOA gene

Purpose

Though stressful life events appear to impact the likelihood and frequency of substance use among adolescents, these effects are often varied and inconsistent. We suggest that the polymorphic MAOA gene may be partially responsible for variable susceptibility to environmental pressures and substance use. More specifically, we hypothesize that adolescents possessing low activity alleles for the MAOA genotype are more likely to respond to stressful life experiences by initiating substance use.

Methods

The genetic subsample of the National Longitudinal Study of Adolescent Health was analyzed (2,574 adolescents) using logistic regression models for each gender. Respondents’ self-reports of eight key stressors were used to create a composite life stress scale which was allowed to interact with a variable that represented the number of low activity MAOA alleles.

Results

For males, a significant interaction emerged between stressful life experiences and the MAOA gene for alcohol (p = .029) and marijuana (p = .039) initiation. For females, the interaction was not significant in each model.

Conclusions

MAOA interacts with life stress to increase the likelihood of substance use initiation for males. Those with a low activity MAOA allele are more likely to initiate substance use than those with a high activity allele when exposed to stressful experiences.     

A biosocial analysis of the sources of missing data in criminological research

Purpose

Failing to deal with missing data patterns effectively may result in biased parameter estimates and ultimately may produce inaccurate results and conclusions. The vast majority of criminological research has addressed this issue with listwise deletion (LD) and multiple imputation (MI) techniques. Identifying the specific covariates that directly contribute to patterns of missingness is highly important in deciding which technique to use. One of the more surprising omissions from the identified list of covariates is the potential role of genetic influences in the development of missingness.

Methods

The current study addresses this gap in the literature by estimating genetic (A), shared environmental (C), and the nonshared environmental (E) influences on missingness across measures of delinquency and self-control within a longitudinal sample of twin and sibling pairs.

Results

The results indicated that genetic influences explain a significant portion of the variance in missing values related to both delinquency and self-control.

Conclusions

Current methodological techniques aimed at addressing missing data should be amended to take genetic influences into account. Such modifications and the implications of the findings for future research are discussed.     

The stability of risk-seeking from adolescence to emerging adulthood

Purpose

The current study examines the stability of the risk-seeking component of self-control using a second-order latent class growth model.

Methods

Longitudinal data from 962 respondents from the NLSY79-Child and Young Adult sample are used to examine the stability of the risk-seeking component of self-control from ages 14 to 23.

Results

Data reveal three trajectories of risk-seeking (low, moderate, and high) that maintain strong relative stability from adolescence through early adulthood. Further, two trajectories of risk-seeking (moderate and high) maintain absolute stability, whereas the low risk-seeking group exhibits statistically significant decreases in risk-seeking over time.

Conclusions

The SOLCGA may provide a stricter test of the stability hypothesis since it accounts for measurement error in the construct prior to estimating the developmental trajectories. The results from the SOLCGA support Gottfredson and Hirschi's hypotheses that self-control will remain stable from adolescence into emerging adulthood.     

Teenage childbirth and young adult criminal convictions: A quasi-experimental study of criminal outcomes for teenage mothers

Purpose

Teenage childbirth is associated with poor psychosocial outcomes for teen mothers. One example is that teen mothers have higher rates of antisocial behavior. The extant research has not been able to determine if teenage motherhood is independently associated with criminal behavior, or if the association is due to selection factors associated with both teenage childbirth and criminal behavior.

Methods

We used longitudinal data from Swedish national registers and sibling-comparisons (both full- and half-siblings) to identify the extent to which there is an independent association between teenage childbirth and mothers’ likelihood of criminal conviction between ages 20-30, or if the association is confounded by familial (including genetic or environmental) factors that make sisters similar.

Results

Women who began childbearing as teenagers were more likely to be convicted of a crime in young adulthood compared to women who delayed childbearing. When sisters were compared, the association between teenage childbirth and criminal convictions disappeared. Multivariate behavior genetic analyses suggest genetic and shared environmental account for the association.

Conclusions

The statistical association between teenage childbirth and early adulthood criminal convictions is confounded by genetic and shared environmental factors that influence both the likelihood of teenage childbirth and risk of early adulthood criminal conviction.     

The genetic and environmental overlap between aggressive and non-aggressive antisocial behavior in children and adolescents using the self-report delinquency interview (SR-DI)

Purpose

This study investigated genetic and environmental commonalities and differences between aggressive and non-aggressive antisocial behavior (ASB) in male and female child and adolescent twins, based on a newly developed self-report questionnaire with good reliability and external validity -- the Self-Report Delinquency Interview (SR-DI).

Methods

Subjects were 780 pairs of twins assessed through laboratory interviews at three time points in a longitudinal study, during which the twins were: (1) ages 9-10 years; (2) age 11-13 years, and (3) age 16-18 years.

Results

Sex differences were repeatedly observed for mean levels of ASB. In addition, diverse change patterns of genetic and environmental emerged, as a function of sex and form of ASB, during the development from childhood to adolescence. Although there was some overlap in etiologies of aggressive and non-aggressive ASB, predominantly in shared environmental factors, their genetic overlap was moderate and the non-shared environmental overlap was low.

Conclusions

Taken together, these results reinforced the importance of differentiating forms of ASB and further investigating sex differences in future research. These results should be considered in future comparisons between youth self-report and parental or teacher report of child and adolescent behavior, and may help elucidate commonalities and differences among informants.     

Evidence for a heritable brain basis to deviance-promoting deficits in self-control

Purpose

Classic criminological theories emphasize the role of impaired self-control in behavioral deviancy. Reduced amplitude of the P300 brain response is reliably observed in individuals with antisocial and substance-related problems, suggesting it may serve as a neurophysiological indicator of deficiencies in self-control that confer liability to deviancy.

Methods

The current study evaluated the role of self-control capacity—operationalized by scores on a scale measure of trait disinhibition—in mediating the relationship between P300 brain response and behavioral deviancy in a sample of adult twins (N = 419) assessed for symptoms of antisocial/addictive disorders and P300 brain response.

Results

As predicted, greater disorder symptoms and higher trait disinhibition scores each predicted smaller P300 amplitude, and trait disinhibition mediated observed relations between antisocial/addictive disorders and P300 response. Further, twin modeling analyses revealed that trait disinhibition scores and disorder symptoms reflected a common genetic liability, and this genetic liability largely accounted for the observed phenotypic relationship between antisocial-addictive problems and P300 brain response.

Conclusions

These results provide further evidence that heritable weaknesses in self-control capacity confer liability to antisocial/addictive outcomes and that P300 brain response indexes this dispositional liability.     

Genetic and environmental influences on the co-occurrence of early academic achievement and externalizing behavior

Purpose

Several studies have observed a relationship between academic achievement and externalizing behaviors, both of which are predictors of delinquency and criminal behavior in adulthood. There is, however, no consensus on an explanation for their co-occurrence. One perspective is that both emerge as a result of a common underlying factor. This study investigates the degree to which the same genetic and environmental factors account for the co-occurrence of these two outcomes.

Methods

The sample consists of twins (N = 360) from the Early Childhood Longitudinal Survey-Kindergarten Class of 1998-1999. Bivariate genetic analyses were conducted to assess the genetic and environmental influence on the relationship between academic achievement and externalizing behaviors during kindergarten.

Results

The covariation was due primarily to common shared environmental factors (55-87%), followed by common genetic (8-44%) and nonshared environmental factors (1-13%).

Conclusions

Both early academic achievement and externalizing behaviors are partially influenced by the same genetic and environmental factors. The large proportion of covariance attributed to shared environmental influences suggests that identifying and targeting shared environmental factors in prevention and intervention strategies may improve both behavior and academic achievement.     

On the relationship of past to future involvement in crime and delinquency: A behavior genetic analysis

Purpose

Criminologists have devoted much attention to identifying the factors that drive stability in antisocial behavior. This body of research has, however, overlooked the contributions of behavior genetic research. This study sought to blend behavior genetics with the different perspectives used by criminologists to explain stability.

Methods

Employing a behavioral genetic research design, the current study analyzed the correlation between adolescent and adulthood crime (a 13 year time span was covered between the two time points) among a sample of sibling pairs drawn from the National Longitudinal Study of Adolescent Health (Add Health).

Results

The findings revealed that genetic factors accounted for nearly all of the stability in offending behavior from adolescence to adulthood. Environmental factors (particularly, of the nonshared variety) accounted for the majority of the changes in offending.

Conclusions

The implications of these results for criminological research and theory are discussed.     

Linking early ADHD to adolescent and early adult outcomes among African Americans

Purpose

The purpose of this study is to propose a mediational model for the mechanisms through which a diagnosis of Attention-Deficit/Hyperactivity Disorder between the ages of 10 and 12 predicts positive and negative early adult outcomes for African Americans.

Methods

The study sample (n = 211) was drawn from the Des Moines, Iowa subsample of the Family and Community Health Study. Participants were first assessed between the ages of 10 and 12, again between the ages of 12 and 18, and finally at 18 to 23.

Results

Findings indicate that a diagnosis of ADHD before age 13 indirectly predicted subsequent exclusionary school discipline and juvenile arrest in adolescence, and both arrests and educational attainment in young adulthood.

Conclusions

These findings offer support for the School to Prison Pipeline model, showing that for some African American children, a childhood diagnosis of ADHD can lead to negative school experiences that result in harsh school-based discipline, which in turn open the door to justice system involvement spanning several developmental stages.     

Sex industry exposure over the life course on the onset and frequency of sex offending

Purpose

Research has examined pornography use on the extent of offending. However, virtually no work has tested whether other sex industry experiences affect sex crime. By extension, the cumulative effect of these exposures is unknown. Social learning theory predicts that exposure should amplify offending. Separately, the developmental perspective highlights that the timing of exposure matters.

Methods

Drawing on retrospective longitudinal data, we first test whether exposure during adolescence is associated with a younger age of onset; we also examine whether adulthood exposure is linked with greater frequency of offending.

Results

Findings indicate that most types of adolescent exposures as well as total exposures were related to an earlier age of onset. Exposure during adulthood was also associated with an overall increase in sex offending, but effects were dependent on “type.”

Conclusion

There are nuances in the effect of sex industry exposure on offending patterns. Implications of results are discussed.     

Aggression and rule-breaking: Heritability and stability of antisocial behavior problems in childhood and adolescence

Purpose

This twin study examined the structure of genetic and environmental influences on aggression and rule-breaking in order to examine change and stability across the span of childhood to mid-adolescence.

Methods

Behavioral assessments were conducted at two time points: age 9–10 years and 14–15 years. Using behavioral genetics biometric modeling, the longitudinal structure of influences was investigated.

Results

Aggression and rule-breaking were found to be influenced by a latent common factor of antisocial behavior (ASB) within each wave of data collection. The variance in the childhood-age common factor of ASB was influenced by 41% genetics, 40% shared environment and 19% nonshared environment. In adolescence, 41% of variance in the common factor were novel and entirely genetic, while the remainder of variance was stable across time. Additionally, both aggression and rule-breaking within each wave were found to have unique influences not common across subscales or across waves, highlighting specificity of genetic and environmental effects on different problem behaviors at both ages.

Conclusions

This research sheds light on the commonality of influences on different forms of antisocial behavior. Future research into interventions for antisocial behavior problems in youth could focus on adolescence-specific environmental influences.     

Adolescent self-image as a mediator between childhood maltreatment and adult sexual offending

Purpose

The link between maltreatment and offending has been well established in the literature, with research examining the etiology of criminal behavior consistently documenting the negative effects of experiencing trauma early in life. Theoretically, ideas behind the cycle of violence hypothesis have greatly contributed to our understanding of this connection; however, there is a lack of understanding of the mechanisms underlying this relationship. We address this research gap by examining the mediating role that adolescent mental health, particularly self-image, plays in the relationship between child maltreatment and subsequent adult sex offending.

Methods

We utilize retrospective data from a sample of incarcerated sex offenders at a maximum security penitentiary located in Canada (N = 565).

Results

Results indicate that poor self-image in adolescence partially mediates the relationship between child maltreatment and the extent of sex offending in adulthood.

Conclusions

Implications for research, theory, and policy are discussed.     

Psychopathic Personality and Negative Parent-to-Child Affect: A Longitudinal Cross-lag Twin Study

Purpose

Previous studies that have explored the relationship between parenting style and children’s antisocial behavior have generally found significant bidirectional effects, whereby parenting behaviors influence their child’s antisocial outcomes, but a child’s behaviors also lead to changes in parenting style.

Methods

The present study investigated the genetic and environmental underpinnings of the longitudinal relationship between negative parent-to-child affect and psychopathic personality in a sample of 1,562 twins. Using a biometrical cross-lag analysis, bidirectional effects were investigated across two waves of assessment when the twins were ages 9-10 and 14-15, utilizing both caregiver and youth self-reports.

Results

Results demonstrated that negative parental affect observed at ages 9-10 influenced the child’s later psychopathic personality at ages 14-15, based on both caregiver and youth self-reports. For these ‘parent-driven effects’, both genetic and non-shared environmental factors were important in the development of later psychopathic personality during adolescence. There were additional ‘child-driven effects’ such that children’s psychopathic personality at ages 9-10 influenced negative parent-to-child affect at ages 14-15, but only within caregiver reports.

Conclusions

Thus, children’s genetically influenced psychopathic personality seemed to evoke later parental negativity at ages 14-15, highlighting the importance of investigating bidirectional effects in parent-child relationships to understand the development of these traits.     

Changes in self-control during adolescence: Investigating the influence of the adolescent peer network

Purpose

Previous research on changes in self-control during adolescence provides evidence of stability for most individuals but change for some, drawing attention to the role of factors that may account for change. We contribute to this area of research by examining the extent to which changes in peer self-control and peer delinquency within peer networks is related to individual self-control during adolescence.

Method

This study uses longitudinal social network data from three waves of the NSCR School Project to construct growth curve models of self-control and MLM models to examine the factors that contribute to changes in self-control.

Results

Growth curve models demonstrate within and between-individual differences in changes to self-control that occur between ages 12-17. Furthermore, multilevel models reveal that changes in levels of self-control and delinquency among respondent's peers are related to between-individual differences and within-individual changes in self-control, net of parental social control.

Conclusions

This study finds that self-control continues to evolve during adolescence as a result of processes that take place within adolescent peer-networks, calling into question Gottfredson and Hirschi's contention that peers are inconsequential for explaining differences in self-control across individuals.     

Foundation for a temperament-based theory of antisocial behavior and criminal justice system involvement

Background

Temperament has been shown to be associated with behavior for millennia but has not been explicitly used in a theory of crime.

Methods

This state-of-the-art review incorporates theory and research from over 300 studies from developmental psychology, psychiatry, genetics, neuroscience, and criminology to introduce a temperament-based theory of antisocial conduct with criminal justice system implications.

Findings

Two temperamental constructs—effortful control and negative emotionality—are significantly predictive of self-regulation deficits and behavioral problems in infancy, in toddlerhood, in childhood, in adolescence, and across adulthood.

Conclusion

Unlike other theories that focus merely on explaining problem behaviors, our temperament approach also explains negative and aversive interactions with criminal justice system practitioners and associated maladjustment or noncompliance with the criminal justice system. A program of research is also offered to examine and test the theory.     

The Consequences of Maladaptive Coping: Integrating General Strain and Self-Control Theories to Specify a Causal Pathway Between Victimization and Offending

Objectives

Drawing from general strain and self-control perspectives, the role of maladaptive coping (i.e., substance use) in the causal pathway between victimization and offending is explored. Specifically, the present study investigates: (1) the extent to which self-control influences substance use in response to victimization, and (2) whether victims with low self-control and who engage in substance use are more likely to commit violent offenses in the future.

Methods

Three waves of panel data from the Gang Resistance Education and Training program are used (N = 1,463), and negative binomial regression models are estimated to explore the interactive effects of low self-control, victimization, and substance use on violent offending.

Results

Victims with low self-control are more likely to engage in substance use post-victimization, and low self-control and substance use are found to exert significant conditional effects on the pathway between victimization and offending. These results remained robust even after controlling for prior violent offending, peer influences, prior substance use, and other forms of offending.

Conclusions

The causal pathway between victimization and offending can be explained by drawing upon key concepts drawn from self-control (i.e., how self-control shapes coping responses) and general strain (i.e., how those responses influence offending above and beyond self-control) theories, indicating that these two perspectives can and should be integrated more explicitly to explain the dynamics of victimization and offending.     

Moving beyond the socialization hypothesis: The effects of maternal smoking during pregnancy on the development of self-control

Purpose

Gottfredson and Hirschi, in A General Theory of Crime, argue that the primary source of self-control is parental socialization. Specifically, parents who fail to supervise their children, to recognize their child's deviant behavior, and to punish such behavior are more likely to raise children with lower levels of self-control. Recent empirical research, however, has broadened the explanatory factors to include sources within schools, neighborhoods, and individual factors as significant contributors to the development of self-control. This study proposes that maternal smoking during pregnancy places additional limits on the development of self-control.

Methods

Using a subsample of the National Longitudinal Study of Youth (N = 542), we provide a comprehensive investigation of the variety of sources of self-control to include both individual and environmental covariates.

Results

Results indicate that maternal smoking during pregnancy significantly impacts the development of self-control net of parental, neighborhood, and school socialization. We also found that individual sources of self-control significantly vary across race and neighborhood context.

Conclusions

The sources of self-control are more complex than socialization from parents, schools, and within neighborhoods occurring in childhood and adolescence.