POLICE VIOLENCE AS A FUNCTION OF COMMUNITY CHARACTERISTICS

The authors find that existing models concerning police violence are not i designed to explain variation among the states in the rates of police use ‘of deadly force. This variation correlates significantly with numerous cultural attributes of the public. The strongest correlates are with the public rates of violence. Therefore, the authors propose to moody existing modeb, and explain the police use of violence as a response to community characteristics. Where public violence is neither normative nor commonly occurrent. the police will be self-restrained. Where the incidence of violence reveals a common means of conflict resolution, the police will adopt its use. Thus, the police use of violence is a culturally determined characteristic and not a police pathology.     

Intravenous amphetamine abuse, primary cerebral mucormycosis, and acquired immunodeficiency

Two intravenous amphetamine abusers had fatal, rapidly progressive cerebral mucormycosis with abscess formation in the presence of absolute lymphopenia. Postmortem examination confirmed the primary nature of the fungal cerebritis, documented by postmortem culture and histology. The clinical and pathologic features of these cases are compared to previously reported occurrences of primary fungal cerebritis (and abscess) among intravenous drug abusers, including cocaine users. Primary fungal cerebritis associated with intravenous abuse of stimulant drugs is discussed as a possible variant of the acquired immunodeficiency syndrome.     

Forensic aspects of digoxin-poisoning: toxicological and morphological findings (author's transl)]

Case report. A 82 year old woman died 80 min after accidental ingestion of 5 mg beta-methyl-digoxin. The autopsy and the histological examination revealed non-specific alterations due to shock and preexisting coronary heart disease. Digoxin levels in various fluids and tissues were estimated by radioimmunoassay: bloodplasma 20--25 ng/ml, liquor 10--13 ng/ml, liver 100--110 ng/g, kidney 130--145 ng/g; the gastric fluid contained 0,6 mg. Forensic aspects of glycosid-intoxication, especially of the varying concentrations in different tissues, are discussed.