帝国主义在东盟地区的利益

近年来,东盟成员国成为美国、日本和西欧等大型帝国主义中心极为关注的对象。东南亚次地区变成了资本主义世界主要工业强国日益增长的政治和经济利益的领域,这些利益的对比关系在一定程度上决定帝国主义“力量中心”在该地区的政策的性质。 这种“利益”取决于一系列重要因素。东南亚次地区优越的地理位置和有利的气候条件有助于相对降低基本建设费用。此外,该地区还是独一无二而丰富的原料的基地,东盟国家是下列产品的大宗生产国和     

苏共二十六大关于成熟社会主义概念的发展

任何一种理论其中包括发达社会主义理论,基本上可归结为二个因素——规律和范畴。因此,发现和研究成熟社会主义的规律以及反映这些客观规律的新概念(新范畴)的科学表达方法乃是成熟社会主义理论的发展趋势。如果个别作者在专著中得出了某些科学结论、原理和概念,而后又反映在党或国家的文件中并加以具体化,也就是说上升为公认的和具有普遍意义的阶段——这是理论发展极重要的因素。对理论研究进行总结并对今后的理论研究提     

评匈牙利经济体制及其发展方向

约亨·贝特肯哈根是德意志经济研究所的研究员,他专门从事对经互会国家的能源经济和对外贸易的研究,主要研究国家是匈牙利和罗马尼亚。     

调动"两大革命"的社会合力--毛泽东与中国近、现代社会变迁概论之三

"思想从来也不能超出旧世界秩序的范围".离开千百万人的革命实践,思想本身"根本不能实现什么东西.为了实现思想,就要有使用实践力量的人"①.引领中国社会变迁的中国共产党人,要实现"两大革命"的历史使命,当然同样不能仅仅停留于对"两大革命"的主观设计,不能停留于它的纯粹"思想"形态,而必须"组织千千万万的民众,调动浩浩荡荡的革命军"②,实际地把"两大革命"胜利完成.事实上,以毛泽东为代表的中国共产党人之所以能够成功引领中国社会变迁,也确实不仅仅是由于它设计了一个"两大革命"的科学蓝图,而且更重要的是由于它同时正确解决了"两大革命"的力量调度这个根本问题,才导演出了"唤起工农千百万,同心干"两大革命"的威武雄壮的活剧.     

Social science research and experimentation in Australian criminal proceedings: prejudicial pre-trial publicity and psychological research

Using the decision of Barr J in Attorney-General (NSW) v John Fairfax Publications [1999] NSWSC 318, the authors analyse the need for external validity and relevance in social science evidence adduced in the courts. They argue in favour of the rigour employed within Barr J's judgment and contend that a constructive legacy of the decision should be a greater sensitisation on the part of researchers to the kinds of factors that can distance experimental scenarios from curial contexts to a point where generalisations from the former become strained and even spurious. A number of these considerations have the potential to be addressed to a significant extent by improved methodologies. However, the authors warn against an excess of purist fervour in demanding complete comparability of scenarios, lest the fruits of social science be denied to the courts and decision-making be adversely affected by the absence of expert insights falling short of complete replication of conditions between experiments and forensic reality.     

STR data for the AmpFlSTR Profiler Plus loci from Greece

Allele frequencies for the nine STRs included in the AmpFlSTR Profiler Plus kit (D3S1358, VWA, FGA, D8S1179, D21S11, D18S51, D5S818, D13S317 and D7S820) were estimated from a sample of 143 unrelated individuals living in different regions of Greece.     

The expression of excitatory amino acid transporter 2 in traumatic brain injury

It is well recognized that glutamate is the major excitatory neurotransmitter, which is removed from the synaptic cleft by excitatory amino acid transporter 2 (EAAT2) located on the perisynaptic astrocytes and that neuronal death has been associated with an increased extracellular glutamate concentration. In this study, we have immunohistochemically demonstrated the expression of EAAT2 protein in the human brain after traumatic brain injury (TBI). The EAAT2 expression patterns can be divided into three types: continuous and highly extensive staining (E); continuous but sporadic staining (M); and sporadic pattern staining (S). In six of the nine short survival cases studied (1 h to 1 day), continuous and highly extensive staining for EAAT2 (E type) was observed in the ipsilateral cerebral cortex. On the other hand, we were able to demonstrate weak staining (S and M types) in 5 of the 7 long survival cases (> or =1 day) and in 12 of the 14 very short survival cases (<1 h) studied. Similar findings were obtained in the contralateral cerebral cortex and also in the ipsilateral hippocampus. In addition, positive staining for glial fibrillary acidic protein was detected around the cerebral contusion, but the EAAT2-positive expression was not observed in the same region for all of the six short and long survival cases (> or =1 h) after TBI. These findings clearly showed the differences in EAAT2 expression in the cerebral cortex according to the survival time and severity of cerebral contusion after TBI. Therefore, we emphasized that EAAT2 might play an important role in contributing to extracellular glutamate concentrations and secondary brain injury after TBI.