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Daisuke Yajima M.D. Ph.D. Keiko Shimizu M.D. Ph.D. Kumiko Oka D.D.S. Ph.D. Masaru Asari Ph.D. Chikatoshi Maseda Ph.D. Katsuhiro Okuda Ph.D. Hiroshi Shiono M.D. Ph.D. Seiji Ohtani LL.B. Katsuhiro Ogawa M.D. Ph.D. 《Journal of forensic sciences》2016,61(Z1):S259-S264
Although Kawasaki disease (KD) is a self‐limiting disease, it may cause sudden cardiac death. Diagnosis of KD is principally based on clinical signs; however, some infant cases do not meet the criteria. Such cases are identified as incomplete KD. The sudden death risk in incomplete KD cases is similar to conventional KD. In our 5‐month‐old case, he had been admitted to a hospital for a fever and suppuration at the site of Bacille de Calmette et Guerin (BCG) vaccination. However, after discharge from the hospital, his C‐reactive protein (CRP) levels declined, he got indisposed and died suddenly. A medico‐legal autopsy revealed myocarditis, coronaritis, platelet‐aggregated emboli in coronary arteries, and myocardial degeneration, suggesting that the fatal myocardial infarction was due to thrombus emboli in the coronary arteries. Forensic pathologists therefore should pay attention to the cardiac pathology originated from incomplete KD as a potential cause in cases of sudden infant death. 相似文献
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Barbara Potocka‐Banaś Ph.D. Tomasz Janus Ph.D. Sławomir Majdanik M.D. Ph.D. Tomasz Banaś LL.M. Teresa Dembińska Ph.D. Krzysztof Borowiak Ph.D. 《Journal of forensic sciences》2017,62(2):553-556
This study presents the fatal case of a young man who was admitted to the ICAU due to sudden cardiac arrest. An interview revealed that the patient had taken some unspecified crystals. From the moment of admission, his condition deteriorated dramatically as a result of increasing circulatory insufficiency. After a few hours, sudden cardiac arrest occurred again and the patient was pronounced dead. In the course of a medicolegal autopsy, samples of biological material were preserved for toxicology tests and histopathological examination. The analysis of samples using the LC‐MS/MS technique revealed the presence of α‐PVP in the following concentrations: blood—174 ng/mL, urine—401 ng/mL, brain—292 ng/g, liver—190 ng/g, kidney—122 ng/g, gastric contents—606 ng/g. The study also presents findings from the parallel histopathological examination. Based on these findings, cardiac arrest secondary to intoxication with alpha‐PVP was determined as the direct cause of the patient's death. 相似文献
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