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Ernesto U. Savona 《European Journal on Criminal Policy and Research》2007,13(3-4):159-160
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In a seminal article, Cox (1990) suggested that electoral systems with larger district magnitudes provide incentives for parties to advocate more extreme policy positions. In this article, we put this proposition to the test. Informed by recent advances in spatial models of party competition, we introduce a design that embeds the effect of electoral rules in the utility function of voters. We then estimate the equilibrium location of parties as the weight voters attach to the expected distribution of seats and votes changes. Our model predicts that electoral rules affect large and small parties in different ways. We find centripetal effects only for parties that are favorably biased by electoral rules. By contrast, smaller parties see their vote share decline and are pushed toward more extreme equilibrium positions. Evidence from 13 parliamentary democracies supports model predictions. Along with testing the incentives provided by electoral rules, results carry implications for the strategies of vote‐maximizing parties and for the role of small parties in multiparty competition. 相似文献
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Fabio De‐Giorgio M.D. Ph.D. Vincenzo M. Grassi M.D. Massimo Miscusi M.D. Ph.D. Cesare Mancuso M.D. Ph.D. Ernesto d'Aloja M.D. Ph.D. Vincenzo L. Pascali M.D. Ph.D. 《Journal of forensic sciences》2013,58(5):1364-1366
This case study reports a fatal subarachnoid hemorrhage (SAH) with concomitant accidental carbon monoxide (CO) exposure in 32‐year‐old man. Autopsy results indicated an antemortem aspiration of smoke, and a massive SAH was identified as the cause of death. Intriguingly, the carboxyhemoglobin level was 30%, suggesting that CO could have played a specific role. Intracranial hemorrhages following CO exposure in brain areas and tissues such as the basal ganglia, globus pallidus, or white matter are rare, but well characterized, whereas SAH related to CO exposure has not been previously described. In this case report, the possible role of CO, either as a primary cause or as a facilitating factor, in the pathogenesis of SAH is discussed. In particular, we propose the hypothesis that the excessive vasodilating effects produced by CO on the cerebral endothelium results in consequential loss of microvascular integrity. 相似文献