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在调整人身侵权债权的各种制度中,为人身侵权债权在破产清偿顺位中找寻优先清偿点是次优制度选择,因为无论人身侵权债权定位在哪一个清偿层面都是后序债权人为破产企业的非法行为买单。为此,在其他法律制度足以保护人身侵权债权人权益的前提下,在破产清偿顺位中为人身侵权债权人设置优先受偿权实属不必要。但是其他法律制度不足以保护人身侵权债权人的权益时,我们可以考虑在破产清偿顺位中采用将人身侵权债权与劳动者债权同序的方式来保护人身侵权债权人的合法权益。 相似文献
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急性及己中毒的实验病理学研究 总被引:2,自引:0,他引:2
目的探讨及己对小鼠毒性作用的病理变化及其毒作用机制。方法应用及己水煎液对小鼠经口急性染毒,进行组织病理学观察血清生化、凝血机能检测。结果及己对小鼠的半数致死量(LD50)为41.12g/kg;染毒小鼠血清ALT和BUN升高;血液血小板计数减少、凝血时间延长;肝、脾、肾器官系数增大;肝、肾、心肌细胞变性、坏死明显,全身诸多器官淤血、出血性改变。结论及己毒作用的主要靶器官或靶组织是肝脏、肾脏、心脏和全身血管。毒作用机制是对线粒体、内质网等膜性结构及体内凝血机制的破坏。 相似文献
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酒精中毒性犯罪及其心理预防 总被引:1,自引:0,他引:1
王敏 《西南政法大学学报》1999,(1)
本文从变态心理学、犯罪学的角度,对酒精中毒与犯罪的关系进行了探讨,根据酒精中毒性犯罪形成、发展的特点,提出了有针对性的三种心理预防方法。 相似文献
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Riza Yilmaz M.D. Eyyüp Yilmaz M.D. Veli Ozdemir M.D. Muhammet Can M.D. Isil Pakis M.D. Ibrahim E. Piskin M.D. Halis Dokgoz M.D. Erdal Ozer M.D. Kemal V. Numanoglu M.D. 《Journal of forensic sciences》2015,60(3):648-652
Yellow phosphorus (YP) is a powerful protoplasmic poison used in the manufacturing of matches, pest poisons, firecrackers, firework cracker, lights for watches, military ammunition, and agriculture fertilizer. YP is extremely flammable and toxic and easily absorbed from the gastrointestinal tract. In this study, we examined childhood deaths from 1997 to 2012 resulting from the ingestion of firecrackers. The patients ranged from 2 to 15 years of age and were admitted to the hospital with a variety of symptoms. Those that presented with nausea, vomiting, and hypotension rapidly deteriorated and entered a coma. An autopsy was performed in all but one of the 16 cases reviewed. Macroscopically, the livers had a yellowish discoloration with petechial bleeding. Histopathologic examination revealed acute toxic hepatitis. In conclusion, these firecrackers are found in corner shops throughout Turkey, may cause death in children with little warning, and should be banned to prevent further deaths. 相似文献
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作者通过大鼠口服敌敌畏和呋喃丹急性中毒实验,利用乙酰胆碱酯酶组织化学技术,研究中毒鼠肋间肌、空肠和大脑乙酰胆碱酯酶活性受抑制和自动复能情况、血液的体外抑制效应以及正常大鼠在室温和冷藏条件下空肠和肋间肌乙酰胆碱酯酶活性的保存时间,探讨利用乙酰胆碱酯酶组织化学方法诊断胆碱酯酶抑制剂中毒的法医学意义,并对大鼠敌敌畏和呋喃丹中毒后上述观察指标的不同特点作了分析比较。 相似文献
28.
Molecular Pathology of Pulmonary Edema in Forensic Autopsy Cases with Special Regard to Fatal Methamphetamine Intoxication 下载免费PDF全文
Rui Zhao Ph.D. M.D. Dong Zhao Ph.D. M.D. Ye Xue M.D. Bao‐Li Zhu Ph.D. M.D. Da‐Wei Guan Ph.D. M.D. Xiao‐Li Xie Ph.D. M.D. Qi Wang Ph.D. M.D. 《Journal of forensic sciences》2016,61(6):1531-1537
Pulmonary edema is a common finding in fatal methamphetamine intoxication. However, the underlying mechanism remains poorly understood. This study investigated the molecular pathology of alveolar damage involving pulmonary edema in forensic autopsy cases. Seven candidate reference genes (RPL13A, YWHAZ, GUSB, SDHA, GAPDH, B2M, and ACTB) were evaluated in the lung by the geNorm module in qBaseplus software. RPL13A, YWHAZ, and GUSB were identified as the most stable reference genes. Using these validated reference genes, intrapulmonary mRNA expressions of matrix metalloproteinases (MMPs), intercellular adhesion molecule‐1 (ICAM‐1), claudin‐5 (CLDN‐5), and aquaporins (AQPs) were examined. Relative mRNA quantification using TaqMan real‐time PCR assay demonstrated higher expressions of all markers except for AQP‐5 in fatal METH intoxication cases. These findings suggested alveolar damage and compensatory response in fatal METH intoxication cases. Systematic analysis of gene expressions using real‐time qPCR is a useful tool in forensic death investigation. 相似文献
29.
Victoria Fischer MD Thomas Kamphausen MD MHBA Andreas Büttner MD Hilke Andresen-Streichert PhD 《Journal of forensic sciences》2023,68(4):1410-1418
A fatal case of 1,4-butanediol (1,4-BD) oral ingestion is reported here, in which a 51-year-old man was found dead in his bed. According to the police report, the deceased was a known drug user. A glass bottle labeled (and later confirmed to be) “Butandiol 1,4” (1,4-BD) was found in the kitchen. Furthermore, the deceased's friend stated that he consumed 1,4-BD on a regular basis. The autopsy and histological examination of postmortem parenchymatous organ specimens did not revealed a clear cause of death. Chemical-toxicological investigations revealed gammahydroxybutyrat (GHB) in body fluids and tissues in the following quantities: femoral blood 390 mg/L, heart blood 420 mg/L, cerebrospinal fluid 420 mg/L, vitreous humor 640 mg/L, urine 1600 mg/L, and head hair 26.7 ng/mg. In addition, 1,4-BD was qualitatively detected in the head hair, urine, stomach contents, and the bottle. No other substances, including alcohol, were detected at pharmacologically relevant concentrations. 1,4-BD is known as precursor substance that is converted in vivo into GHB. In the synoptic assessment of toxicological findings, the police investigations and having excluded other causes of death, a lethal GHB-intoxication following ingestion of 1,4-BD, can be assumed in this case. Fatal intoxications with 1,4-BD have seldom been reported due to a very rapid conversion to GHB and, among other things, non-specific symptoms after ingestion. This case report aims to give an overview to the published of fatal 1,4-BD-intoxications and to discuss the problems associated with detection of 1,4-BD in (postmortem) specimens. 相似文献
30.
Mattias Kettner M.D. Frank Ramsthaler M.D. Christian Juhnke Dipl.Ing. Roman Bux M.D. Peter Schmidt M.D. 《Journal of forensic sciences》2013,58(2):556-558
Carbon dioxide (CO2) is an odorless constituent of air. Higher concentrations can be detected in geothermal and automotive emissions, fermentation, and sublimation of dry ice. An unskilled worker entered a fermentation tank to clean it, which had not been done for about 5 months allowing for high concentrations of CO2 to build up. A second worker entered the tank to rescue the first one. Shortly after both were found the first worker was rescued directly whereas the tank had to be rotated to pull the second worker out. Cardiopulmonary resuscitation was successful only for the first worker. Medico‐legal autopsy showed bruises, hematoma, myocardial hemorrhage, and edema of the lungs. The right lung was vacuum degassed in an argon atmosphere and quadrupole‐mass‐spectrometry showed an elevated CO2 content in lung gases. Thus, CO2 intoxication/asphyxia in a vitiated atmosphere due to fermentation of wine mash was established as the cause of death. 相似文献